新型冠状病毒感染后肺纤维化的机制及治疗进展_Chinese Journal of Respiratory and Critical Care Medicine

Authors：

季辰菲 , 陶媛 ,  吉宁飞

南京医科大学第一附属医院呼吸与危重症医学科（江苏南京 210029）;

Corresponding author：

吉宁飞, Email: jiningfei@163.com

Keywords：

DOI：

10.7507/1671-6205.202504075

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Citation： 季辰菲, 陶媛, 吉宁飞. 新型冠状病毒感染后肺纤维化的机制及治疗进展. Chinese Journal of Respiratory and Critical Care Medicine, 2025, 24(10): 739-744. doi: 10.7507/1671-6205.202504075 Copy

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5. Hoffmann M, Kleine-Weber H, Schroeder S, et al. SARS-CoV-2 cell entry depends on ACE2 and TMPRSS2 and is blocked by a clinically proven protease inhibitor. Cell, 2020, 181(2): 271-280. e8.
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11. Yuen CK, Lam J , Wong WM, et al. SARS-CoV-2 nsp13, nsp14, nsp15 and orf6 function as potent interferon antagonists. Emerg Microbes Infect, 2020, 9(1): 1418-1428.
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14. Wendisch D, Dietrich O, Mari T, et al. SARS-CoV-2 infection triggers profibrotic macrophage responses and lung fibrosis. Cell, 2021, 184(26): 6243-6261. e27.
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新型冠状病毒感染后肺纤维化的机制及治疗进展

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