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find Keyword "糖尿病肾病" 48 results
  • 糖尿病肾病与炎症致病作用及治疗研究进展

    糖尿病肾病(diabetic nephropathy,DN)是糖尿病的重要微血管病变之一,其发病虽与高血糖明确相关,但仍有研究显示严格控制血糖以及应用血管紧张素转换酶抑制剂或血管紧张素Ⅱ受体拮抗剂等药物仅能部分延缓DN进展,绝大部分患者最终缓慢进展至终末期肾衰竭,新的发病机制亟待探索。最近研究揭示炎症机制是DN发生发展的关键因素,本文就DN与炎症发病机制,致病作用及治疗的研究进展作相关阐述。

    Release date:2016-09-08 09:49 Export PDF Favorites Scan
  • The Clinical Therapeutic Effect of Astragalus Mongholicus (AM) in Treatment of Diabetic Nephropathy

    目的:观察黄芪注射液治疗糖尿病肾病的临床疗效。 方法:将116例糖尿病肾病患者随机分为治疗组和对照组,治疗组在对照组的基础上同时使用黄芪注射液,观察治疗后4周24小时尿蛋白定量、血肌酐、尿素氮、血尿β2微球蛋白、甘油三酯、总胆固醇、高密度脂蛋白胆固醇、低密度脂蛋白胆固醇、血尿酸等变化.结果:治疗组治疗后24小时尿蛋白定量、血尿酸均有不同程度的改善,与治疗前比较Plt;0.05,治疗组与对照组比较Plt;0.05。而血尿β2微球蛋白、血胆固醇无明显变化。结论:黄芪注射液对糖尿病肾病有较好的疗效。

    Release date:2016-09-08 10:04 Export PDF Favorites Scan
  • 胰岛素联合罗格列酮治疗早期糖尿病肾病

    【摘要】 目的 观察胰岛素联合罗格列酮治疗早期糖尿病肾病的疗效。方法 2008年3月-2009年2月,将61例符合早期糖尿病肾病的患者随机分为治疗组和对照组。对照组30例予糖尿病基础治疗,治疗组31例加用胰岛素和罗格列酮。治疗观察6个月。比较治疗前后两组空腹血糖(FBG)、糖化血红蛋白(HbA1c)、胰岛素抵抗指数(HomaIR)、尿白蛋白排泄率(UAER)等变化情况。结果 治疗组在治疗后FBG、HbA1c、HomaIR、UAER均较治疗前明显改善,差异有统计学意义(Plt;0.05),且FBG、HbA1c、UAER的下降较对照组明显(Plt;0.05)。 结论 胰岛素联合罗格列酮可有效调控早期糖尿病肾病患者的血糖,改善胰岛素抵抗,减少尿蛋白排泄,保护肾功能。

    Release date:2016-09-08 09:31 Export PDF Favorites Scan
  • Clinical Observation on Suxuetong Injection with ACEI/ARB for Early Diabetic Nephropathy

    目的:观察采用疏血通注射液联合ACEI/ARB治疗早期糖尿病肾病(DN)的疗效。方法:将78例2型DN患者随机分为对照组(ACEI/ARB)和治疗组(ACEI/ARB+疏血通注射液),疗程4周。比较两组治疗前和治疗后尿微量白蛋白(mAlb),Scr、BUN等指标的变化。结果:(1)治疗后治疗组和对照组尿白蛋白均显著下降(Plt;0.01,Plt;0.05),治疗组比对照组下降更为明显(Plt;0.05)。(2)治疗后两组血浆白蛋白均增加(Plt;0.01),治疗组与对照组治疗后比较无明显差异(Pgt;0.05)。(3)治疗后两组Scr、BUN、TC、TG和血钾均无明显变化。结论:联合应用疏血通注射液能有效减少早期DN患者的蛋白尿,改善肾功能。

    Release date:2016-09-08 10:01 Export PDF Favorites Scan
  • Progress in diagnosis and treatment of diabetic kidney disease

    Diabetic kidney disease (DKD) is a major complication of diabetes mellitus. One third of patients with advanced diabetes mellitus can develop to uremia, which seriously endangers people’s health. In recent years, with the improvement of people’s living standards and the increasing incidence of diabetes, it has become the main cause of end stage renal disease in China. Over the past two decades, the understanding of diagnosis and treatment of DKD has been improved, such as putting forward the new concept of normoalbuminuric DKD and developing a variety of new anti-diabetic drugs. However, at present, the basic strategies of DKD treatment are still lifestyle modification, glucose control, blood pressure control and lipid control.

    Release date:2019-08-15 01:18 Export PDF Favorites Scan
  • Research progress of metabonomics in early diagnosis of diabetic kidney disease

    Diabetic kidney disease, as a common complication of diabetes, is one of the main causes of end-stage renal disease. Because of the rapid progress of its course and the limited means of treatment, it is of great clinical significance to seek biomarkers from early diagnosis for the treatment of diabetic kidney disease. At present, there are limited methods for early diagnosis of diabetic kidney disease. As a widely used research method, metabonomics can detect metabolites in diseases and provide biomarkers for disease diagnosis and prognosis. This article summarizes the changes of amino acids, lipids, organic acids and other metabolites in blood or urine of patients with diabetic kidney disease.

    Release date:2023-04-24 08:49 Export PDF Favorites Scan
  • 乙酰肝素酶与糖尿病肾病

    【摘要】 乙酰肝素酶(heparanase,HPA)是目前发现的哺乳动物细胞中唯一能切割细胞外基质中硫酸肝素蛋白多糖侧链的内源性糖苷酶。因其可促进肿瘤细胞的浸润和转移,还可以促进肿瘤细胞生长和微血管形成,而被视为是抗肿瘤、抗炎症的理想靶点。研究表明HPA可以降解肾小球基底膜硫酸乙酰肝素多糖侧链,造成肾小球基底膜选择性滤过蛋白质功能下降,与糖尿病肾病(diabetic nephropathy,DN)蛋白尿的产生密切相关。HPA活性的高低对糖尿病肾病的研究有着重要的意义。现将HPA与DN之间的研究进展作一综述。

    Release date:2016-09-08 09:26 Export PDF Favorites Scan
  • Kaishi Injection for Diabetic Nephropathy: A Systematic Review

    Objective To carry out Meta analyses about the published literature that concerns Kaishi injection curing diabetic nephropathy, and to evaluate the efficacy and safety of Kaishi injection for diabetic nephropathy. Methods We searched the following databases: PubMed (1995 to 2010), EMCC (1995 to 2010), CBM (1995 to 2010), CNKI (1995 to 2010), and VIP (1989 to 2010) to collect randomized controlled trials (RCTs) of Kaishi injection curing diabetic nephropathy. The selection of studies, assessment of methodological quality and data extraction were performed independently by two reviewers. According to predefined inclusion and exclusion criteria Cochrane systematic review methods, the methodological quality assessment was undertaken, and meta-analyses were performed by using The Cochrane Collaboration’s RevMan 4.2.8 software. Evolution index were included: UAER, Scr, BUN, and 24 hours urinary protein.Results The literature included 19 RCTs with a total of 1 153 cases. Among them, 594 cases belonged to the treatment group and the control group included 559 ones. The studies of baseline data were comparable, and all reported that there were random methods but did not mention blinding and allocation concealment. Only one mentioned references to a listof random numbers by random grouping. The results of meta-analyses indicated that Kaishi injection was superior to routine treatment in decreasing UAER [WMD= – 77.86, 95%CI (– 85.64, – 70.08)], Scr [WMD= – 3.14, 95%CI (– 5.30, – 0.98)], BUN [WMD= – 0.71, 95%CI (– 1.13, – 0.29)], and 24 hours urinary protein [WMD= – 0.56, 95%CI (– 0.79, – 0.33)]. Conclusion The treatment of the diabetic nephropathy of Kaishi injection is superior to the conventional therapy. However, because of few high quality literature and limited sample size, further study is needed.

    Release date:2016-09-07 11:23 Export PDF Favorites Scan
  • RhoA/Rho相关卷曲螺旋形成的蛋白激酶信号通路与慢性肾脏疾病

    【摘要】 RhoA/Rho相关卷曲螺旋形成的蛋白激酶(ROCK)信号通路是细胞内重要的信号转导通路,在慢性肾脏疾病的进展中起重要作用。肾间质纤维化是各种慢性肾脏疾病发展到终末期肾衰竭的共同途径;糖尿病肾病是继发性慢性肾脏疾病的主要病因。现就RhoA/ROCK信号通路在肾间质纤维化和糖尿病肾病发病机制中的作用作一综述。

    Release date:2016-09-08 09:26 Export PDF Favorites Scan
  • Protective Effect of Roux-en-Y Gastric Bypass Surgery on Early Damage of Renal Tissue in Type 2 Diabetes Mellitus Rats

    ObjectiveTo investigate the protective effect of Roux-en-Y gastric bypass surgery on early damage of renal tissue in type 2 diabetes mellitus rats, and explore the mechanism of the protective effects. MethodsDiabetes mellitus animal models were induced by intraperitoneal injection of streptozotocin (STZ, 35 mg /kg) and a high-fat diet.Diabetic rats were divided into three groups randomly (digital table method): diabetes control group (n=8), sham operation group (n=8), and Roux-en-Y gastric bypass group (n=14).Another 8 normal SD rats as the normal control group.The fasting blood glucose, serum total cholesterol (TC), triglyceride (TG), and free fatty acid (FFA) were measured before operation and in 8 weeks after operation; plasma BUN and Cr were measured respectively before operation and in 4 and 8 weeks after operation in each group rats, 24 h urine microalbumin and urine 8-hydroxydeoxyguanosine were measured respectively before operation and in 8 weeks after operation in each group rats.Renal pathological changes were observed and the indexes of kidney hypertrophy, the mean glomerular area (MGA), and the mean glomerular volume (MGV) were analyzed in 8 weeks after operation.The expressions of fibronectin, typeⅣcollagen (CoⅣ), transforming growth factor-β1 (TGF-β1), intercellular adhesion molecule-1(ICAM-1), nicotinamide adenine dinucleotide phosphate oxidase 4 (NOX4), and Bcl-2 protein in renal tissues were investigated by immunohistochemical staining. ResultsRoux-en-Y gastric bypass surgery could reduce the blood glucose, blood lipid, MGA, MGV, and the index of kidney hypertrophy of diabetic rats significantly (P < 0.05), improved renal pathological morphology and kidney function (P < 0.05), reduced the protein expressions of fibronectin and CoⅣ, decreased the protein expressions of TGF-β1, ICAM-1, and NOX4, and increased the protein expression of Bcl-2. ConclusionRoux-en-Y gastric bypass surgery can improve kidney function and the pathological damage of diabetes rats, its mechanism may be related to inhibition the protein expressions of TGF-β1, ICAM-1, and NOX4, and increase the protein expression of Bcl-2.

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